EXAMINATION OF A BIOPSYCHOSOCIAL MODEL FOR THE RELATIONSHIP BETWEEN POSTTRAUMATIC STRESS DISORDER AND CHRONIC PAIN

First Name: 
Anna
Last Name: 
Cassel
Field of Study: 
Psychology
Keywords: 
Posttraumatic stress disorder, PTSD, chronic pain, musculoskeletal, pain

eXAMINATION OF A BIOPSYCHOSOCIAL MODEL FOR THE RELATIONSHIP BETWEEN POSTTRAUMATIC STRESS DISORDER

AND CHRONIC PAIN

By Anna G. Cassel

Thesis Advisor: Sandra T. Sigmon, Ph.D.

An Abstract of the Thesis Presented in Partial

Fulfillment of the Requirements for the

Degree of Doctor of Philosophy

(in Psychology)

August, 2010

 

            High rates of comorbidity have been reported between PTSD and musculoskeletal pain (e.g., Asmundson & Hadjistavropolous, 2006; Asmundson et al., 1998).  Comorbid PTSD and chronic pain have been associated with elevated levels of affective distress, greater perceptions of pain, interference in daily activities, and high rates of disability (Otis et al., 2003; Sherman et al., 2000).  Overall, comorbid conditions of PTSD and chronic pain are associated with large personal costs for the individual and economic costs for society.

            The triple vulnerability model was originally proposed to account for anxiety symptoms in general, and it was later applied to the specific development of PTSD (Barlow, 2000; Barlow, 2002; Keane & Barlow, 2002).  Otis and colleagues (2003) further proposed that the triple vulnerability model may account for the relationship between PTSD and chronic pain.  According to the triple vulnerability model, individuals must present with a generalized biological, generalized psychological, and a specific psychological vulnerability for either of these conditions to develop (Keane & Barlow, 2002; Otis et al., 2003).

 

            In the current study, aspects of the triple vulnerability model were examined within the following groups of women:women who have PTSD without chronic pain (n = 11), women who have musculoskeletal pain without PTSD (n = 10), women with both PTSD and musculoskeletal pain (n = 10), and women without PTSD and chronic pain (n = 15).  Cortisol reactivity and anxious mood were assessed before and after the Trier Social Stress Task (TSST).  Participants also completed questionnaires to assess for other potential indicators of the triple vulnerability model.

            Results indicate that: 1) the roles of generalized biological, generalized psychological, and specific psychological vulnerabilities toward developing PTSD were supported; 2) limited findings supported the potential role of these vulnerabilities toward developing chronic pain; however, results of these measures were not similar to that of PTSD (e.g., family history of chronic pain); 3) it is not thought that PTSD and chronic pain are associated with the same vulnerabilities; 4) having a diagnosis of PTSD and chronic pain was associated with an increase in symptoms across many measures utilized in the current study.